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Neil HALLIDAY, Speciality Registrar | Cited by 1,904 | of Royal Free London NHS Foundation Trust, London (Royal Free) | Read 60 publications | Contact Neil HALLIDAY
Neil Halliday Senior Lecturer Nottingham School of Art & Design Email: neil.halliday@ntu.ac.uk; Phone: +44 (0)115 941 8418 Contact us +44 (0)115 848 2999 ...
4 gen 2018 · Cytotoxic T-lymphocyte antigen 4 (CTLA-4) (CD152) and CD28 are homologous receptors expressed by both CD4 + and CD8 + T cells, which mediate opposing functions in T-cell activation. Both receptors share a pair of ligands expressed on the surface of antigen-presenting cells (APCs).
- Behzad Rowshanravan, Neil Halliday, David M. Sansom
- 2018
8 dic 2020 · Abstract. CD80 and CD86 are expressed on antigen presenting cells and are required to engage their shared receptor, CD28, for the costimulation of CD4 T cells. It is unclear why two stimulatory ligands with overlapping roles have evolved. CD80 and CD86 also bind the regulatory molecule CTLA-4.
- Neil Halliday, Cayman Williams, Alan Kennedy, Erin Waters, Anne M Pesenacker, Blagoje Soskic, Claudi...
- 2020
Abstract. Background: Current therapeutic options for autoimmune hepatitis (AIH) are limited by adverse events associated with corticosteroids and thiopurines and the limited evidence base for second- and third-line treatment options.
- Neil Halliday, Neil Halliday, Jessica Katharine Dyson, Jessica Katharine Dyson, Douglas Thorburn, Do...
- 2020
Neil Halliday is a full-time Senior Lecturer teaching across all three years on the BA Graphic design course at NTU.
Background. Data show that patients with autoimmune hepatitis have significantly reduced quality-of-life and. that corticosteroids carry marked side effects. . Aims. This study explored patients’ experiences of autoimmune hepatitis and its treatments; key aspects.